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Foals may also show unusual behavior, such as head-pressing, licking at the stall walls, and startling at sounds or touch.

In severe cases, a dummy foal may become recumbent and experience seizures. The underlying cause is believed to be lack of oxygen in the perinatal period.

According to Petroski, mare owners should consider natural variations in gestational length when attempting to classify a poor-doing foal. This is why veterinarians recommend attended foalings, potential problems identified swiftly, and all foals examined by their veterinarian within hours of birth.

EO-3 is a potent, effective source of marine-derived long-chain omega-3 fatty acids , which have shown multiple benefits for mares and foals, such as improving follicular growth, enhancing embryo quality, improving endometrial scores, and boosting colostrum quality.

Nano-E , a liquid, natural-source vitamin E product, uses nanodispersion technology to boost absorption rates. These foals are also predisposed to angular limb deformities and must be closely monitored for development of this problem as they mature.

Postmature foals may be affected by flexural contracture deformities , most likely due to decreased intrauterine movement as they increase in size.

The overall prognosis for premature, dysmature, and postmature foals remains fair to good with intensive care and attention to detail.

Many foals survive and become productive athletes. Complications associated with sepsis and musculoskeletal abnormalities are the most significant indicators of poor athletic outcome.

Neonatal Encephalopathy Etiology A wide spectrum of clinical signs is associated with neonatal encephalopathy, ranging from mild depression with loss of suckle reflux to grand mal seizures.

Affected foals are typically healthy at birth but show signs of CNS abnormalities within a few hours.

However, the timing of onset of clinical signs varies; some foals are obviously abnormal at birth, and some do not show clinical signs until 24 hours of age.

Neonatal encephalopathy is commonly associated with adverse peripartum events, including dystocia, placentitis, twinning, and premature placental separation.

However, some foals have no known evidence for the cause of the hypoxic event, suggesting that unrecognized in utero hypoxia occurred.

Seizures must be controlled, because they increase cerebral oxygen consumption by 5-fold. Diazepam 0. Sepsis in foals can be quite subtle initially, and the onset of clinical signs is variable depending on the pathogen involved and the immune status of the foal.

Common pathogens include gram-negative bacteria, although gram-positive infections have been identified. Failure of passive transfer of immunity can contribute to development of sepsis in foals at risk.

Other risk factors for development of sepsis include any adverse event at the time of birth, maternal illness, or any abnormalities in the foal.

Although the umbilicus is frequently implicated as a major portal of entry for infectious organisms, the GI tract may be the primary site of entry.

Other portals of entry include the respiratory tract and wounds. Survival rates of foals treated for sepsis have improved, but infection must be recognized early for the possibility of a good outcome.

The pathogen involved can also affect survival. Gram-negative species are more commonly diagnosed, but gram-positive septicemia is being recognized more frequently, and multiple organisms may be involved.

It is important to identify the organism early in the course of the disease. Blood cultures should be obtained, as well as samples from synovial fluid, CNS, peritoneal fluid, urine, and tracheal aspirates if localized signs are present.

Until antimicrobial sensitivity patterns for the pathogen involved are obtained, broad-spectrum antimicrobial therapy should be started.

Therapeutic drug monitoring may be needed to ensure adequate drug levels. Failure of passive transfer should be treated, if present, with hyperimmune plasma.

Mechanical ventilation may be necessary in cases of severe respiratory involvement seen with acute lung injury or acute respiratory distress syndrome.

If the foal is hypotensive, pressor agents or inotropes may be administered by constant-rate infusion. NSAIDs are used by some practitioners, as are corticosteroids, in specific circumstances.

Use of these drugs should be judicious, because they may have severe negative consequences, including, but not limited to, renal failure and gastric or duodenal ulceration.

Antiulcer medications are controversial, because critically ill, recumbent foals typically have an alkaline gastric pH.

These medications may be more useful once the foal is ambulatory. Supportive care is important in treatment of septic foals.

Foals should be kept warm and dry and turned at 2-hour intervals if recumbent. Every attempt should be made to keep the foal sternal to improve respiratory function and reduce lung atelectasis.

Feeding septic foals can be a challenge if GI function is abnormal; total parenteral nutrition may be needed. If at all possible, foals should be weighed daily and blood glucose levels monitored frequently.

Some foals become persistently hyperglycemic on low glucose infusion rates. These foals may benefit from constant-rate infusions of insulin.

Recumbent foals must be examined frequently for decubital ulcers, corneal ulcers, and for heat and swelling associated with the joints and physes.

Physical therapy or passive range of motion exercises should be provided. The prognosis for foals in the early stages of sepsis is fair to good.

Once the disease has progressed to septic shock, the prognosis becomes less favorable, although short-term survival rates are similar to those seen in human patients.

Long-term survival and athletic outcomes are fair. Racing-breed foals that make it to the track perform similarly to their age-matched siblings.

Premature, dysmature, and postmature foals appear similar clinically in many regards and require aggressive nursing care for survival. Issues such as neonatal encephalopathy and sepsis may be concurrent.

Neonatal encephalopathy is managed by methods to support cerebral perfusion, control edema, and prevent seizures. Secondary respiratory depression may require mechanical ventilation.

Sepsis may result from in utero or extrauterine sources and may be related to failure of transfer of passive immunity. Significant time and effort is required in the short term for treatment of septic foals.

However, outcomes can be good if treated early, before localization of the infection. Also see Pet Health content regarding emergency care for horses.

From developing new therapies that treat and prevent disease to helping people in need, we are committed to improving health and well-being around the world.

The Merck Veterinary Manual was first published in as a service to the community. This site complies with the HONcode standard for trustworthy health information: verify here.

Common Veterinary Topics. Videos Figures Images Quizzes. Initial Assessment. Dystocia and Resuscitation.

Prematurity, Dysmaturity, and Postmaturity. Etiology Diagnosis Treatment. Neonatal Encephalopathy. Etiology Treatment. Etiology Signs Diagnosis Treatment.

Key Points. For More Information. Equine Emergency Medicine. Test your knowledge. Colic abdominal pain is a common equine emergency. Which of the following is a common procedure that can easily be performed by a veterinarian in the field to aid diagnosis and provide therapy?

More Content. Clinical Evaluation Early recognition of abnormalities is of utmost importance for successful management of critically ill foals.

Basic Life Support If the foal does not breathe or move spontaneously to right itself within seconds of birth, tactile stimulation is necessary eg, drying with a towel.

Physical examination. Oxygen therapy potentially with mechanical ventilation. Etiology A wide spectrum of clinical signs is associated with neonatal encephalopathy, ranging from mild depression with loss of suckle reflux to grand mal seizures.

Septic arthritis is usually due to bacteria in the blood stream infecting the joint. Left untreated, even if only for 24 to 48 hours, it can rapidly lead to irreparable damage to the joint cartilage and permanent consequences for soundness.

Therefore, if your foal becomes lame it is wise to seek veterinary attention as soon as possible as early identification and treatment of septic arthritis can prevent permanent damage occurring.

Diarrhoea in foals is a very common issue and can vary from being a mild transient problem to being a serious life threatening condition.

This diarrhoea is actually not caused by the mare being in season but is due to the foal's gut adjusting and establishing the normal gut flora for digestion.

Foals should remain bright, continue to drink well and not have a temperature. If they show any of these signs then a more serious infectious diarrhoea might be involved.

Foals can get severe diarrhoea caused by bacteria types such as Salmonella, E. There are also viral diarrhoeas, the most common being caused by Rotavirus.

With these more serious conditions, the diarrhoea is very severe, smells strongly and may be projectile and even bloody. Without treatment, the foals can become severely dehydrated, hypoglycaemic low blood glucose from not suckling and also prone to developing other infections for example septicaemia due to the compromised state.

Some diarrhoeas we might be able to manage at your home with oral medications, other more serious cases may require hospitalisation for intravenous fluids and intensive care nursing.

Respiratory Conditions Respiratory disease is a major type of illness in the foal. It can potentially be life threatening and can also hold long term consequences for future performance.

Other respiratory diseases of foals include bacterial infections such as Rhodococcus equi and Streptococcus equi, viral infections such as Equine herpesvirus and Equine influenza and parasitic conditions.

Some of these illnesses will require hospitalisation and intensive care, however, many can be managed at home with appropriate care and medication.

Foal Diseases and Syndromes. Prematurity and Dysmaturity Premature foals are those who have been born between to days gestation pregnancy duration.

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This condition is also referred to as barkers, sleepers, wanderers, neonatal maladjustment syndrome, hypoxic-ischemic encephalopathy, neonatal encephalopathy, or perinatal asphyxia syndrome.

Foals may also show unusual behavior, such as head-pressing, licking at the stall walls, and startling at sounds or touch.

In severe cases, a dummy foal may become recumbent and experience seizures. The underlying cause is believed to be lack of oxygen in the perinatal period.

According to Petroski, mare owners should consider natural variations in gestational length when attempting to classify a poor-doing foal.

This is why veterinarians recommend attended foalings, potential problems identified swiftly, and all foals examined by their veterinarian within hours of birth.

EO-3 is a potent, effective source of marine-derived long-chain omega-3 fatty acids , which have shown multiple benefits for mares and foals, such as improving follicular growth, enhancing embryo quality, improving endometrial scores, and boosting colostrum quality.

Nano-E , a liquid, natural-source vitamin E product, uses nanodispersion technology to boost absorption rates.

Subscribe to Equinews and get the latest equine nutrition and health news delivered to your inbox. Sign up for free now! Search Library Entire Site.

Foals with rib fractures should be placed in lateral recumbency, with the fractured ribs down for compressions. After resuscitation, ultrasound can be used to identify rib fractures that have escaped detection by palpation or new fractures caused by compressions.

Ultrasound is the most sensitive diagnostic tool to identify rib fractures. Epinephrine remains the drug of choice at a dosage of 0. If given through the nasotracheal tube, the dosage should be 0.

Epinephrine can be repeated every 4 minutes during compressions, coinciding with the second assessments between rounds of compressions. Vasopressin 0.

Atropine is not recommended in bradycardic newborn foals, because the bradycardia is usually secondary to hypoxia.

Atropine can also increase myocardial oxygen debt if hypoxia is not corrected. Doxapram is not recommended for resuscitation of newborns, because it does not reverse secondary apnea.

Immediately after birth, the foal must adapt to independent thermoregulation. In response to the catecholamine surge associated with birth, uncoupling of oxidative phosphorylation occurs within the mitochondria, releasing energy as heat.

This non-shivering thermogenesis is impaired in newborns undergoing hypoxia or asphyxiation and in those ill at birth.

Human infants born to mothers sedated by benzodiazepines are similarly affected, a consideration in the choice of sedative and preanesthetic medications in mares with dystocia or undergoing cesarean section.

In addition to non-shivering thermogenesis, thermoregulation in the healthy foal is supported by a high metabolic rate, a thick hair coat, fat stores, and the ability to shiver within minutes of birth.

Heat losses by convection, radiation, and evaporation are quite high in most areas where foals are delivered, resuscitated, and managed, and care must be taken to ensure that cold stress is minimized in newborn and critically ill foals.

The foal should be dried and placed on dry bedding once resuscitation is compete. Supplemental heat in the form of radiant lamps or warm air-circulating blankets may be required.

Fluid therapy should be used conservatively in postpartum resuscitation. The neonate is typically not volume depleted unless excessive hemorrhage has occurred.

Some compromised newborns are actually hypervolemic. Because the renal function of the equine neonate is substantially different from that of adult horses, fluid therapy cannot simply be scaled down.

Indications for this shock bolus include poor mentation, poorly palpable peripheral pulses, and development of cold distal extremities compatible with hemorrhagic shock.

The foal should be assessed after the initial bolus, with additional boluses up to three administered as needed. This therapy is indicated to maintain blood glucose levels, resolve metabolic acidosis, and support cardiac output, because myocardial oxygen stores have likely been depleted.

Premature foals are small, with a fine, silky hair coat, generalized muscle weakness, joint and tendon laxity, incomplete cuboidal bone ossification, a domed forehead, and floppy ears.

Foals born post-term, but small, are termed dysmature. These foals may also exhibit the characteristic signs of prematurity. A postmature foal is a post-term foal that has a normal axial skeletal size but is thin to emaciated.

The hair coat is generally long, and the teeth may have erupted in utero. Postmature foals are usually healthy foals that have been retained too long in utero, perhaps due to an abnormal signaling of readiness for birth.

Postmature foals become more abnormal the longer they are maintained in utero, and they may suffer from placental insufficiency.

Prematurity, dysmaturity, and postmaturity may all be associated with high-risk pregnancy. Postmature foals are commonly born to mares ingesting endophyte-infested fescue.

Iatrogenic causes include early elective induction of labor based on inaccurate breeding dates or interpretation of late-term colic or uterine bleeding as ineffective labor.

Most often, the cause is idiopathic. Even if undetermined, the cause may continue to affect the foal after birth.

All body systems may be affected by prematurity, dysmaturity, and postmaturity, and thorough evaluation is necessary.

Respiratory failure is common in these foals and is related to immaturity of the respiratory tract, poor control of respiratory vessel tone, and weak respiratory muscles, combined with poorly compliant lungs and a greatly compliant chest wall.

It is usually not due to a surfactant deficiency. Most foals require oxygen supplementation and positional support for optimal oxygenation and ventilation.

Some may require mechanical ventilation. These foals also require cardiovascular support but are frequently unresponsive to commonly used pressors and inotropes, including dopamine , dobutamine , epinephrine , and vasopressin.

Careful use of these drugs and judicious IV fluid therapy are necessary. Renal function, reflected in low urine output, is often initially poor because of a delay in making the transition from fetal to neonatal glomerular filtration rates.

The delay can be due to true failure of transition or secondary to a hypoxic or ischemic insult. Fluid therapy should be used cautiously in these cases; an initial fluid restriction may be required to avoid fluid overload.

The Holliday-Segar formula is recommended to prevent excessive fluid administration. Many premature, dysmature, or postmature foals have suffered a hypoxic insult and present with all of the disorders associated with perinatal asphyxia syndrome, inducing neonatal encephalopathy see below.

Treatment is similar to that of term foals with these problems. These foals are also predisposed to secondary bacterial infections and must be examined frequently for signs consistent with early sepsis or nosocomial infection.

The GI system of these foals is not usually functionally mature because of a primary lack of maturity or secondary to hypoxia.

Dysmotility and varying degrees of necrotizing enterocolitis are common, as are hyperglycemia and hypoglycemia. Hyperglycemia is generally related to stress, increased levels of circulating catecholamines, and a rapid progression to gluconeogenesis, whereas hypoglycemia is associated with diminished glycogen stores, the inability to engage gluconeogenesis, sepsis, and hypoxic damage.

Endocrine function may be immature, particularly regarding the hypothalamic-pituitary-adrenal axis, and contributes to metabolic derangements.

If possible, enteral feeding should be delayed until metabolic and cardiorespiratory parameters are stable and parenteral nutrition is provided.

When enteral feeding is initiated, small volumes should be provided first and slowly increased throughout several days.

Musculoskeletal problems are frequent, particularly in premature foals, and include significant flexor laxity, periarticular ligament laxity, and decreased muscle tone.

Premature foals frequently exhibit flexor laxity combined with decreased cuboidal bone ossification that predisposes them to crush injury of the carpal and tarsal bones if weight bearing is not strictly controlled.

Physical therapy, in the form of assisted standing and controlled exercise, is indicated in the management of these problems; however, care should be taken to ensure that the foal does not become fatigued and stand in abnormal positions.

Splints and casts only increase laxity in the limbs, although light bandages over the fetlock may be necessary to prevent injury if flexor laxity is severe.

Glue-on shoes may help improve the weight-bearing axis. If tube casts are used, they should not extend below the fetlock to minimize complications of joint laxity, and they should be changed regularly to prevent sores.

These foals are also predisposed to angular limb deformities and must be closely monitored for development of this problem as they mature.

Postmature foals may be affected by flexural contracture deformities , most likely due to decreased intrauterine movement as they increase in size.

The overall prognosis for premature, dysmature, and postmature foals remains fair to good with intensive care and attention to detail.

Many foals survive and become productive athletes. Complications associated with sepsis and musculoskeletal abnormalities are the most significant indicators of poor athletic outcome.

Neonatal Encephalopathy Etiology A wide spectrum of clinical signs is associated with neonatal encephalopathy, ranging from mild depression with loss of suckle reflux to grand mal seizures.

Affected foals are typically healthy at birth but show signs of CNS abnormalities within a few hours. However, the timing of onset of clinical signs varies; some foals are obviously abnormal at birth, and some do not show clinical signs until 24 hours of age.

Neonatal encephalopathy is commonly associated with adverse peripartum events, including dystocia, placentitis, twinning, and premature placental separation.

However, some foals have no known evidence for the cause of the hypoxic event, suggesting that unrecognized in utero hypoxia occurred. Seizures must be controlled, because they increase cerebral oxygen consumption by 5-fold.

Diazepam 0. Sepsis in foals can be quite subtle initially, and the onset of clinical signs is variable depending on the pathogen involved and the immune status of the foal.

Common pathogens include gram-negative bacteria, although gram-positive infections have been identified.

Failure of passive transfer of immunity can contribute to development of sepsis in foals at risk.

Other risk factors for development of sepsis include any adverse event at the time of birth, maternal illness, or any abnormalities in the foal.

Although the umbilicus is frequently implicated as a major portal of entry for infectious organisms, the GI tract may be the primary site of entry.

Other portals of entry include the respiratory tract and wounds. Survival rates of foals treated for sepsis have improved, but infection must be recognized early for the possibility of a good outcome.

The pathogen involved can also affect survival. Gram-negative species are more commonly diagnosed, but gram-positive septicemia is being recognized more frequently, and multiple organisms may be involved.

It is important to identify the organism early in the course of the disease. Blood cultures should be obtained, as well as samples from synovial fluid, CNS, peritoneal fluid, urine, and tracheal aspirates if localized signs are present.

Until antimicrobial sensitivity patterns for the pathogen involved are obtained, broad-spectrum antimicrobial therapy should be started.

Therapeutic drug monitoring may be needed to ensure adequate drug levels. Failure of passive transfer should be treated, if present, with hyperimmune plasma.

Mechanical ventilation may be necessary in cases of severe respiratory involvement seen with acute lung injury or acute respiratory distress syndrome.

If the foal is hypotensive, pressor agents or inotropes may be administered by constant-rate infusion. NSAIDs are used by some practitioners, as are corticosteroids, in specific circumstances.

Use of these drugs should be judicious, because they may have severe negative consequences, including, but not limited to, renal failure and gastric or duodenal ulceration.

Antiulcer medications are controversial, because critically ill, recumbent foals typically have an alkaline gastric pH.

These medications may be more useful once the foal is ambulatory. Supportive care is important in treatment of septic foals.

Foals should be kept warm and dry and turned at 2-hour intervals if recumbent. Every attempt should be made to keep the foal sternal to improve respiratory function and reduce lung atelectasis.

Feeding septic foals can be a challenge if GI function is abnormal; total parenteral nutrition may be needed.

If at all possible, foals should be weighed daily and blood glucose levels monitored frequently. Some foals become persistently hyperglycemic on low glucose infusion rates.

These foals may benefit from constant-rate infusions of insulin. Recumbent foals must be examined frequently for decubital ulcers, corneal ulcers, and for heat and swelling associated with the joints and physes.

Physical therapy or passive range of motion exercises should be provided. The prognosis for foals in the early stages of sepsis is fair to good. Once the disease has progressed to septic shock, the prognosis becomes less favorable, although short-term survival rates are similar to those seen in human patients.

Long-term survival and athletic outcomes are fair. Racing-breed foals that make it to the track perform similarly to their age-matched siblings.

Premature, dysmature, and postmature foals appear similar clinically in many regards and require aggressive nursing care for survival.

Issues such as neonatal encephalopathy and sepsis may be concurrent. Neonatal encephalopathy is managed by methods to support cerebral perfusion, control edema, and prevent seizures.

Secondary respiratory depression may require mechanical ventilation. Sepsis may result from in utero or extrauterine sources and may be related to failure of transfer of passive immunity.

Significant time and effort is required in the short term for treatment of septic foals. However, outcomes can be good if treated early, before localization of the infection.

Also see Pet Health content regarding emergency care for horses.

Persistent bradycardia is an indication for rapid intervention. Dismature Treatment. Absence of a menace reflex should not be considered diagnostic of visual deficits in newborn foals. Septicaemia This is the name given to a condition where pathogenic capable of causing illness bacteria are Sexy mexican pornstars in the foal's bloodstream and it most commonly affects foals less than a week of age. There is an extreme variation in the severity of the clinical signs foals show with this syndrome and therefore the prognosis for these cases Dismature enormously and is extremely difficult to predict. Sexo en la granja foals may be affected by flexural contracture deformitiesmost likely due to decreased intrauterine movement as they increase in size. Sepsis in Pornhud lesbian can be quite Lebo sex initially, and Nikki blond anal onset of clinical signs is variable depending on the pathogen involved and the immune status of the foal. Xxx crossdresser given through the Big tit dance tube, the dosage should be 0. Dismature

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